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penemuan baru siklus tidur menjelaskan mengapa diet lemak selama kehamilan membuat anak-anak obesitas
Hubungan antara tidur dan obesitas diambil sebagai penelitian baru yang menunjukkan bahwa apa yang ibu Anda makan saat hamil dapat membuat Anda gemuk atau kelebihan berat badan dengan mengubah fungsi gen ( perubahan epigenetik ) yang mengatur ritme sirkadian ....read more
New sleep cycle
discovery explains why fatty diets during pregnancy make kids obese
Date:
November 23, 2010
Source:
Federation of American
Societies for Experimental Biology
Summary:
The link between
sleeping and obesity is drawn tighter as a new research shows that what your
mother ate when she was pregnant may make you obese or overweight by altering
the function of genes (epigenetic changes) that regulate circadian rhythm.
..............
the link between
sleeping and obesity is drawn tighter as a new research published online in the FASEB
Journal study shows
that what your mother ate when she was pregnant may make you obese or
overweight by altering the function of genes (epigenetic changes) that regulate
circadian rhythm. In the report, pregnant primate females consuming a high-fat
diet altered the function of fetal genes that regulate circadian rhythm
(including appetite and food intake) during development. The offspring also had
non-alcoholic fatty liver disease.
"It is our hope that our studies will continue to guide research aimed
at understanding the pivotal role that maternal health plays in guiding the
health of the next generation," said Kjersti Aagaard-Tillery, M.D., Ph.D.,
a researcher involved in the work from the Department of Obstetrics and
Gynecology at Baylor College of Medicine in Houston. "We are enthusiastic
that our research will give hope that even small changes, such as improving
one's diet in pregnancy and during breastfeeding, will translate into a lower
chance for obesity in our next generation."
To make this discovery, Aagaard-Tillery and colleagues studied three groups
of Japanese macaque primates. One group was fed a 12 percent fat diet (the
control group). The second group was fed a 35 percent fat or high-fat diet and
the third group was fed the high-fat diet for up to five years and then
switched back to the control diet. Each group maintained their specific diet
prior to conception and throughout pregnancy. Offspring from the high-fat group
developed non-alcoholic fatty liver disease; experienced changes in histones
(the core set of proteins which DNA wrap around), and had altered metabolic
profiles and circadian rhythms. Results also showed that the genes in the fetal
liver, which are responsible for orchestrating circadian rhythms with appetite
and food intake, are altered in offspring from the mothers on the high-fat
diet. Specifically, one of these genes, called Npas2, is a key regulator of the
circadian system and is itself regulated by changes in the fetal histone code.
Scientists found that improving the diet, either for the pregnant or
breastfeeding mother, or for the infant after birth, helps to partially restore
the circadian machinery back to normal, possibly lessening the risk of
childhood diseases related to obesity.
"We've recently published a number of studies in the FASEB
Journal showing that what a mother eats affects the weight of her
children for their entire lives," said Gerald Weissmann, M.D., Editor-in-Chief
of the FASEB Journal. "Now, we know why, and what a wake-up
call for pregnant women! The mother's diet during pregnancy affects their
children's sleep machinery via genetic machinery that controls the sleep cycle.
Children are literally forced to sleep in the proverbial bed their mothers have
made."
Story Source:
The above story is based on materials provided by Federation of American Societies
for Experimental Biology. Note: Materials may be edited for content and length.
Journal Reference:
1. M. Suter, P. Bocock, L. Showalter, M.
Hu, C. Shope, R. McKnight, K. Grove, R. Lane, K. Aagaard-Tillery. Epigenomics:
maternal high-fat diet exposure in utero disrupts peripheral circadian gene
expression in nonhuman primates.The FASEB Journal, 2010; DOI: 10.1096/fj.10-172080
