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Alkohol membuat Anda sejenak bahagia tapi tidak memberikan kepuasan lebih---T-REC semarang--komunitas reptil-semarang--KSE-komunitas satwa eksotik--berita artikel terkait tentang alkohol

Alkohol membuat Anda sejenak bahagia tapi tidak memberikan kepuasan lebih
Date:
May 4, 2016
Source:
University of Kent
Summary:
Penelitian menunjukkan orang yang sesaat bahagia saat minum alkohol - tapi
untuk waktu yang lebih lama , minum lebih banyak tidak membuat mereka lebih puas dengan kehidupan
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Penelitian menunjukkan orang yang sesaat bahagia saat minum alkohol - tapi untuk  waktu yang lebih lama , minum lebih banyak tidak membuat mereka lebih puas dengan kehidupan .
 
Penelitian , yang dipimpin oleh seorang ahli kebijakan sosial di University of Kent , juga menemukan bahwa orang yang bermasalah dengan  minum kurang puas dengan kehidupan .

Meskipun efek alkohol pada kebahagiaan sering dibahas selama debat tentang kebijakan alkohol dan regulasi , itu jarang menjadi subyek studi akademis yang serius . Sebaliknya , pemerintah  hanya menggunakan asumsi ekonom bahwa setiap orang selalu bertindak rasional dan dalam kepentingan terbaik mereka - bahkan ketika mereka mabuk atau kecanduan alkohol .

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EXOTIC PETS COMMUNITY-- INDONESIA


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Alcohol makes you momentarily happier but not more satisfied
Date:
May 4, 2016
Source:
University of Kent
Summary:
Research suggests people are momentarily happier when drinking alcohol -- but that over longer periods, drinking more does not make them more satisfied with life.
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Research suggests people are momentarily happier when drinking alcohol -- but that over longer periods, drinking more does not make them more satisfied with life.
The research, led by a social policy expert at the University of Kent, also found that people who developed drinking problems were less satisfied with life.
Although the effect of alcohol on happiness is often discussed during debates about alcohol policy and regulation, it has rarely been the subject of serious academic study. Instead, governments have simply used the economist assumption that everyone always acts rationally and in their best interests -- even when they are drunk or addicted to alcohol.
The study considered how people's happiness and drinking change alongside each other over a period of time. The authors, Dr Ben Baumberg Geiger of the University's School of Social Policy, Sociology and Social Research, and Dr George MacKerron of the University of Sussex, made use of both an iPhone-based app and a traditional cohort study to generate the findings.
The results suggested that, after making allowances for other factors such as illness that can effect wellbeing, there was no connection between people's drinking and their happiness over a period of time. The exception to this was in situations where alcohol became a problem, leading to reduced feelings of wellbeing.
Both studies took into account other possible explanations for the relationship between alcohol and happiness, although the authors concede that being absolutely sure that alcohol is causing momentary happiness is difficult. They also acknowledge that those involved in the studies are not representative of the whole population. The first study involved iPhone users, who tend to be young and wealthy, while the second study looks only at 30-42 year olds.
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studi memperhitungkan penjelasan lain yang mungkin untuk hubungan antara alkohol dan kebahagiaan , meskipun penulis mengakui bahwa menjadi benar-benar yakin bahwa alkohol menyebabkan kebahagiaan sesaat adalah  sulit . Mereka juga mengakui bahwa mereka yang terlibat dalam studi tidak mewakili seluruh populasi . Penelitian ini melibatkan pertama  Para pengguna iPhone , yang cenderung muda dan kaya, sedangkan studi kedua terlihat hanya pada usia 30-42 tahun .

But the study does offer at least some robust evidence when policymakers previously had nothing but 'pub talk' to rely on, say the paper's authors. They hope the research will help policymakers properly take happiness into account when doing cost-benefit analyses of alcohol regulation -- and therefore make better, more transparent decisions about which policies will benefit the population and which won't.


Story Source:
The above post is reprinted from materials provided byUniversity of Kent. The original item was written by Martin Herrema. Note: Materials may be edited for content and length.


Journal Reference:
1.      Ben Baumberg Geiger, George MacKerron. Can alcohol make you happy? A subjective wellbeing approach.Social Science & Medicine, 2016; 156: 184 DOI:10.1016/j.socscimed.2016.03.034
Sumber :


Gen terkait Ketergantungan alkohol dengan neurotransmiter--T-REC--komunitas reptil-semarang--KSE--komunitas satwa eksotik

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More info :
minat gabung : ( menerima keanggotaan seluruh kota dan daerah di Indonesia )
08995557626
..................................
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Visit Our Community and Joint W/ Us....Welcome All Over The World
 KSE = KOMUNITAS SATWA EKSOTIK

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KAMI ADA DI TIAP KOTA DI INDONESIA 

GABUNG......... ( menerima keanggotaan seluruh kota dan daerah di Indonesia )
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Gen terkait Ketergantungan alkohol  dengan neurotransmiter


Para ilmuwan telah memecahkan misteri mengapa jalur signaling tertentu dapat dikaitkan dengan ketergantungan alkohol. Penelitian baru menunjukkan gen, Nf1, mengatur asam gamma - aminobutirat (GABA), neurotransmitter yang menurunkan kecemasan dan meningkatkan perasaan relaksasi..........



Alcohol-dependence gene linked to neurotransmitter
Date:
August 27, 2014
Source:
Scripps Research Institute
Summary:
Scientists have solved the mystery of why a specific signaling pathway can be associated with alcohol dependence. The new research shows the gene, Nf1, regulates gamma-aminobutyric acid (GABA), a neurotransmitter that lowers anxiety and increases relaxation feelings
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Scientists at The Scripps Research Institute (TSRI) have solved the mystery of why a specific signaling pathway can be associated with alcohol dependence.
This signaling pathway is regulated by a gene, called neurofibromatosis type 1 (Nf1), which TSRI scientists found is linked with excessive drinking in mice. The new research shows Nf1 regulates gamma-aminobutyric acid (GABA), a neurotransmitter that lowers anxiety and increases feelings of relaxation.
"This novel and seminal study provides insights into the cellular mechanisms of alcohol dependence," said TSRI Associate Professor Marisa Roberto, a co-author of the paper. "Importantly, the study also offers a correlation between rodent and human data."
In addition to showing that Nf1 is key to the regulation of the GABA, the research, which was published recently in the journal Biological Psychiatry, shows that variations in the human version of the Nf1 gene are linked to alcohol-dependence risk and severity in patients.
Pietro Paolo Sanna, associate professor at TSRI and the study's corresponding author, was optimistic about the long-term clinical implications of the work. "A better understanding of the molecular processes involved in the transition to alcohol dependence will foster novel strategies for prevention and therapy," he said.
A Genetic Culprit
Researchers have long sought a gene or genes that might be responsible for risk and severity of alcohol dependence. "Despite a significant genetic contribution to alcohol dependence, few risk genes have been identified to date, and their mechanisms of action are generally poorly understood," said TSRI Staff Scientist Vez Repunte-Canonigo, co-first author of the paper with TSRI Research Associate Melissa Herman.
This research showed that Nf1 is one of those rare risk genes, but the TSRI researchers weren't sure exactly how Nf1 affected the brain. The TSRI research team suspected that Nf1 might be relevant to alcohol-related GABA activity in an area of the brain called the central amygdala, which is important in decision-making and stress- and addiction-related processes.
"As GABA release in the central amygdala has been shown to be critical in the transition from recreational drinking to alcohol dependence, we thought that Nf1 regulation of GABA release might be relevant to alcohol consumption," said Herman.
The team tested several behavioral models, including a model in which mice escalate alcohol drinking after repeated withdrawal periods, to study the effects of partially deleting Nf1. In this experiment, which simulated the transition to excessive drinking that is associated with alcohol dependence in humans, they found that mice with functional Nf1 genes steadily increased their ethanol intake starting after just one episode of withdrawal. Conversely, mice with a partially deleted Nf1 gene showed no increase in alcohol consumption.
Investigating further, the researchers found that in mice with partially deleted Nf1 genes, alcohol consumption did not further increase GABA release in the central amygdala. In contrast, in mice with functional Nf1 genes, alcohol consumption resulted in an increase in central amygdala GABA.
In the second part of the study, a collaboration with a distinguished group of geneticists at various U.S. institutions, the team analyzed data on human variations of the Nf1 gene from about 9,000 people. The results showed an association between the gene and alcohol-dependence risk and severity.
The team sees the new findings as "pieces to the puzzle." Sanna believes future research should focus on exactly how Nf1 regulates the GABA system and how gene expression may be altered during early development.

Story Source:
The above story is based on materials provided by Scripps Research Institute. Note: Materials may be edited for content and length.

Journal Reference:
  1. Vez Repunte-Canonigo, Melissa Herman, Tomoya Kawamura, Henry R. Kranzler, Richard Sherva, Joel Gelernter, Lindsay A. Farrer, Marisa Roberto, Pietro Paolo Sanna. Nf1 regulates alcohol dependence-associated excessive drinking and GABA release in the central amygdala in mice, and is associated with alcohol dependence in humans.. Biological Psychiatry, 2014; DOI: 10.1016/j.biopsych.2014.07.031



















 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
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