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Obat yang digunakan untuk mengobati kanker tampaknya mempertajam memori

Petunjuk untuk menjaga sel-sel otak hidup pada mereka dengan Alzheimer

Date:

October 2, 2015

Source:

Rutgers University

Summary:

Sebuah obat yang  sekarang digunakan untuk mengobati kanker mungkin membuatnya lebih mudah untuk belajar bahasa , mempertajam memori dan membantu mereka dengan demensia dan penyakit Alzheimer dengan rewiring otak dan menjaga neuron hidup . Penelitian baru menemukan bahwa obat - RGFP966 - diberikan kepada tikus membuat mereka lebih terbiasa dengan apa yang mereka dengar , mampu mempertahankan dan mengingat informasi lebih lanjut, dan mengembangkan hubungan baru yang memungkinkan memori  ini akan ditransmisikan antara sel-sel otak 

.......... Penelitian baru Rutgers dipublikasikan dalam Journal of Neurosciencefound bahwa obat - RGFP966 - diberikan kepada tikus membuat mereka lebih terbiasa dengan apa yang mereka dengar , mampu mempertahankan dan mengingat informasi lebih lanjut , dan mengembangkan hubungan baru yang memungkinkan kenangan ini akan ditransmisikan antara sel-sel otak ....more

Drug used to
treat cancer appears to sharpen memory

Clues to keeping brain cells alive in those with Alzheimer's

Date:

October 2, 2015

Source:

Rutgers University

Summary:

A drug now being used to treat cancer might make it easier to learn a
language, sharpen memory and help those with dementia and Alzheimer's disease
by rewiring the brain and keeping neurons alive. New research found that a drug
-- RGFP966 -- administered to rats made them more attuned to what they were
hearing, able to retain and remember more information, and develop new
connections that allowed these memories to be transmitted between brain cells.

.....................

Can you imagine a drug that would make it easier to learn a language,
sharpen your memory and help those with dementia and Alzheimer's disease by
rewiring the brain and keeping neurons alive?

New Rutgers research published in the Journal of Neurosciencefound
that a drug -- RGFP966 -- administered to rats made them more attuned to what
they were hearing, able to retain and remember more information, and develop
new connections that allowed these memories to be transmitted between brain
cells.

"Memory-making in neurological conditions like Alzheimer's disease is
often poor or absent altogether once a person is in the advanced stages of the
disease," said Kasia M. Bieszczad, lead author and assistant professor in
Behavioral and Systems Neuroscience in the Department of Psychology. "This
drug could rescue the ability to make new memories that are rich in detail and
content, even in the worst case scenarios."

What happens with dementias such as Alzheimer's is that brain cells shrink
and die because the synapses that transfer information from one neuron to
another are no longer strong and stable. There is no therapeutic treatment
available that reverses this situation.

The drug being tested in this animal study is among a class known as HDAC
inhibitors -- now being used in cancer therapies to stop the activation of
genes that turn normal cells into cancerous ones. In the brain, the drug makes
the neurons more plastic, better able to make connections and create positive
changes that enhance memory. Researchers found that laboratory rats, taught to
listen to a certain sound in order to receive a reward, and given the drug
after training, remembered what they learned and responded correctly to the
tone at a greater rate than those not given the drug.

Scientists also found that the rodents were more "tuned in" to
the relevant acoustic signals they heard during their training -- an important
finding Bieszczad said because setting up the brain to better process and store
significant sounds is critical to human speech and language.

"People learning to speak again after a disease or injury as well as
those undergoing cochlear implantation to reverse previous deafness, may be
helped by this type of therapeutic treatment in the future," said
Bieszczad "The application could even extend to people with delayed
language learning abilities or people trying to learn a second language."

This hypersensitivity in processing auditory information enabled the
neurons to reorganize and create new pathways -- allowing more of the
information they learned to become a long-term memory, said Bieszczad who
collaborated with colleagues in the Department of Neurobiology and Behavior at
the University of California Irvine.

"People normally remember an experience with limited detail -- not
everything we see, hear and feel is remembered," she said. "What has
happened here is that memory becomes closer to a snapshot of the actual
experience instead of being sparse, limited or inaccurate."










Story Source:

The above post is reprinted from materials provided byRutgers
University. Note: Materials may be edited for content and length.










Journal Reference:

1.   
K. M. Bieszczad, K. Bechay, J. R. Rusche, V. Jacques, S. Kudugunti, W.
Miao, N. M. Weinberger, J. L. McGaugh, M. A. Wood. Histone Deacetylase
Inhibition via RGFP966 Releases the Brakes on Sensory Cortical Plasticity and
the Specificity of Memory Formation. Journal of Neuroscience,
2015; 35 (38): 13124 DOI:10.1523/JNEUROSCI.0914-15.2015

http://www.sciencedaily.com/releases/2015/10/151002113548.htm

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